In cooperation with TargetMol, we provide inhibitors for multiple research areas. Please check our catalogue and website for more informations. Custom compound library is also available.

From small molecule compounds to large molecule products, such as antibodies, peptides, and proteins; from bioproducts to genetically engineered products; from catalogue products to custom services, we EJOLIFE are specialized in providing complete solutions for your research with competitive price. Your success is our success.

In cooperation with TargetMol, we provide compound various compound libraries, including bioactive compound libraries, natural product libraries, fragment libraries, scaffold & scaffold-based libraries, and custom library upon request. Please check our catalogue.

The main group of enzymes responsible for the collagen and other protein degradation in extracellular matrix (ECM) are matrix metalloproteinases (MMPs) [1]. Matrix metalloproteinases (MMPs) are a large family of calcium-dependent zinc-containing endopeptidases, which are responsible for the tissue remodeling and degradation of the extracellular matrix (ECM), including collagens, elastins, gelatin, matrix glycoproteins, and proteoglycan [2]. MMP family can be divided into six groups: collagenases, gelatinases, stromelysins, matrilysins, membrane-type MMPs, and other non-classified MMPs [3]. The MMPs are involved in many biologic processes, such as, tissue repair and remodulation, cellular differentiation, embryogenesis, morphogenesis , cell mobility, angiogenesis, cell proliferation and migration, wound healing, apoptosis. The deregulation of MMPs activity leads to development of numerous diseases [1, 4].

In the past decades, Neuroscience has emerged as a key area of life science. Neuroscience includes but not limited to neural development and cell lineage determination, functional regulation of neural system, synaptic plasticity, neuro- endocrine system, neurotransmitter metabolism and regulation, neural vesicle trafficking, neurotransmission and channels, neurotrophin and neuro-protective mechanism, molecular mechanism of neurodegeneration and neural injury. We have a range of research tools for neuroscience.